Browse95 live movements — 115 models — 108 quiz cases — 362 teaching concepts
Playpress ▶ to animate — adjust Amplitude / Frequency / Speed sliders
Readclinical pearl below the eyes — use Differentiate / Cases for deeper learning
LEARN:
Eye Movement Visual Fixation — selected
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WAVEFORM
MUSCLES
Clinical Pearl
CLINICAL NOTES

Differential Comparison

Select two movements to compare side-by-side with live animation and differential analysis.

Case Practice

Clinical vignette + eye movement — name the diagnosis. Select an answer then reveal explanation.

Press "Start Quiz" above to begin.

🔬 INO Severity Grading

Select INO from the movement list, then choose a grade below. Grade 1 = subtle lag; Grade 4 = complete adduction failure.

Grade 3 (default): Marked adduction lag — eye reaches <25% of target with obvious dissociated nystagmus in abducting eye. Switch to ino movement to see effect.

Signal Trace

Real-time position trace — Blue: O.D. horizontal  |  Orange: O.S. horizontal  |  Red: vertical  |  Purple: torsion OD

Anatomy / Exam

6 extraocular muscles — right eye, frontal view. MR/LR = rectus pair; SR/IR = vertical rectus; SO/IO = obliques.

🔍 HINTS Protocol

Select HINTS Exam from the movement list. Step through Head Impulse → Nystagmus → Test of Skew → Hearing (S+) to distinguish central (stroke) from peripheral (benign) vertigo. All 4 positive = STROKE.

Examination Step
Pattern
⚠️ CENTRAL PATTERN (Stroke until proven otherwise)
Normal HIT + Direction-changing nystagmus + Skew + Sudden hearing loss = INFARCT.
Action: Admit, urgent MRI, do NOT discharge.

🔁 BPPV Fatigability

Select BPPV Posterior from the list. Each repeated Dix-Hallpike reduces amplitude by ×0.7 (canalolithiasis fatigues). Cupulolithiasis does NOT fatigue.

Nystagmus Amplitude 100% (untested)

completed Dix-Hallpike cycles

↪ Returning to upright — press again to trigger next cycle

📏 Alexander's Law

Select Alexander's Law Demo. Watch nystagmus intensity rise in right gaze (toward fast phase) and fall in left gaze (away from fast phase).

◀ LEFT CENTER GAZE RIGHT ▶
Nystagmus Intensity 80%
Primary Position: Baseline nystagmus — 80% intensity.

Fast phase = right. Right gaze = 150% · Center = 80% · Left gaze = 40%

🌀 Inner Ear Canal Anatomy

Shows which semicircular canal is affected. Select any BPPV variant from the list — the active canal highlights and debris animates.

🔴 Canalolithiasis
Free debris
🟠 Cupulolithiasis
Fixed on cupula
⬜ Inactive
Not involved

🧠 Brainstem Pathway Map

Interactive brainstem localisation. Hover a structure to see its function. Click to load the eye movement it produces when damaged.

🔴 PPRF
Horizontal gaze
🟡 MLF
INO pathway
🟢 Cerebellum
Gaze stability

🎯 Cerebellar Localisation Game

Score: 0 / 0

Observe the eye movement in the simulator above. Identify which cerebellar structure is lesioned.

Press Start to begin.

🌡️ COWS Caloric Step-Through

Cold Opposite · Warm Same — step through all 4 caloric combinations with physiology.

Click a step above to begin.
Mnemonic: COLD = OPPOSITE  ·  WARM = SAME

⚡ Wernicke's Encephalopathy Progression

Auto-cycles through 3 stages (~12 sec each). Select Wernicke's Encephalopathy from the list.

⚠️ MEDICAL EMERGENCY — IV Thiamine 500 mg TDS × 3 days before any glucose
Stage 1 — Nystagmus Auto-cycling
Gaze-evoked nystagmus + upbeat nystagmus in primary position. Reversible with IV thiamine.

🚩 Central vs Peripheral — Red Flag Flashcards

Score: 0 / 0

Observe the eye movement in the simulator. Decide: is this pattern Central (brainstem/cerebellar) or Peripheral (labyrinth/vestibular nerve)?

Press Start to begin.

🌀 Head-Shaking Nystagmus Test

Select Head-Shaking Nystagmus Test. Phase 1 (10s): horizontal head oscillation. Phase 2: post-shake nystagmus with exponential decay. Choose variant below.

Direction Variant
Clinical pearl: Perverted HSN (vertical nystagmus after horizontal head shake) is a central sign — brainstem or cerebellum. Always examine in Frenzel goggles.

📳 Vibration-Induced Nystagmus (VIN)

Select VIN + affected ear (sidebar). 100Hz mastoid vibration — nystagmus ONLY during stimulus. SPV ≥2°/s = abnormal.

Nystagmus Type
Horizontal: 91% VN · 94% Ramsay Hunt · toward healthy ear
Vertical (upbeat): SCDS / superior canal — not UVL alone
VIN tests high-frequency VOR — can be positive when caloric is entirely normal. Stops immediately when vibration ends.

💨 Hyperventilation-Induced Nystagmus (HVIN)

Select HVIN + affected ear (sidebar). Auto-cycles: 30s hyperventilation → nystagmus builds → 15s rest.

Variant
Pearl: HVIN toward affected side = acoustic neuroma. Tumor-compressed nerve excited by alkalosis.
Builds 10–20s, peaks 25–30s. Peripheral loss: contralesional. Acoustic neuroma: paradoxical ipsilesional.

🔊 Superior Canal Dehiscence (SCDS)

Select SCDS from dropdown. No nystagmus at rest. Activate sound or pressure stimulus below.

Tullio Phenomenon
Sound-triggered nystagmus
Hennebert Sign
Pressure-triggered nystagmus
Direction: Vertical-torsional in superior canal plane. Upper pole toward affected ear.
Diagnosis: CT temporal bone 0.5mm cuts (not MRI).

😴 Myasthenia Gravis — Fatigability

Select Myasthenia Gravis — Ocular Fatigability. Eyes in sustained upgaze — watch progressive drift down over 60 seconds as NMJ fatigues.

NMJ Fatigue 0%
Key features: Pupil sparing · Fatigability · No single nerve pattern · Variable muscles
Distinguishes from: CN III palsy (pupil involved), INO (fixed pattern)

🎯 Optokinetic Nystagmus (OKN)

Select Optokinetic Reflex (OKN). OKN is a visual reflex — full amplitude is present when the patient sees the rotating drum. Set drum rotation direction below.

Drum Rotation Direction
Clinical rule: Slow phase follows the drum; fast phase (named direction) resets opposite. OKN asymmetry (weaker in one direction) indicates ipsilateral parietal or occipital lesion.